theoretical comparability or divergence

Do family-design and case-control GWASs arrive at the same answers? Any papers discussing actual/theoretical comparability or divergence?

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For diseases that are heritable, unaffected parents in a family-based design seem like they might have a higher SNP-mediated burden for disease as compare with a control randomly drawn from the relevant ancestral population.    In fact, some parents may have a mild form of the phenotype.

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